Honors College Senior Thesis Presentations

Characterization of Pancreatic Development in CRISPR/Cas9 Zebrafish Model

Presenter Information

Caleb HarsinFollow

Academic Level at Time of Presentation

Senior

Major

Chemistry/Pre-medicine

Minor

Cell Biology

List all Project Mentors & Advisor(s)

Dr. Gary ZeRuth

Presentation Format

Oral Presentation - Murray State Access only

Abstract/Description

Gli-similar 3 (Glis3) has been implicated in several human pathologies including neonatal diabetes, congenital hypothyroidism, and polycystic kidney disease. Numerous genome-wide association studies (GWAS) have additionally identified Glis3 as a risk locus for the development of type 2 diabetes mellitus. To elucidate Glis3 gene function, a CRISPR-mediated knockout line of zebrafish was developed by deleting a segment of the Glis3 coding region resulting in a non-functional protein product. At select time points in development, the resulting embryos were analyzed via fluorescence imaging to determine the effect of the Glis3 knockout on embryonic pancreatic development.

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Honors College Senior Thesis Presentations

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Characterization of Pancreatic Development in CRISPR/Cas9 Zebrafish Model

Gli-similar 3 (Glis3) has been implicated in several human pathologies including neonatal diabetes, congenital hypothyroidism, and polycystic kidney disease. Numerous genome-wide association studies (GWAS) have additionally identified Glis3 as a risk locus for the development of type 2 diabetes mellitus. To elucidate Glis3 gene function, a CRISPR-mediated knockout line of zebrafish was developed by deleting a segment of the Glis3 coding region resulting in a non-functional protein product. At select time points in development, the resulting embryos were analyzed via fluorescence imaging to determine the effect of the Glis3 knockout on embryonic pancreatic development.