University of Kentucky

Drug Use and Brain Injury in Kentucky Acute Care Facilities in 2020

Grade Level at Time of Presentation

Junior

Major

Public Health

Institution

University of Kentucky

KY House District #

7

KY Senate District #

8

Department

College of Public Health, Kentucky Injury Research and Prevention Center

Abstract

Introduction: Individuals who overdose may acquire a brain injury due to hypoxia, and individuals with traumatic brain injury are at greater risk for substance misuse. The purpose of this project was to determine if there is cooccurrence of drug use and overdose and brain injury.

Methods: Using the 2020 Kentucky Outpatient Service and Inpatient Hospitalization Database, encounters for traumatic brain injury (TBI), acquired, non-traumatic brain injury (NTBI), substance use disorder, and overdose were identified using the International Classification of Disease, 10th Edition – Clinical Management. To collect this data, ICD-10-CM codes T36.X-T50.X were used to code for drug use and F11-F19 to code for drug disorder. Coding for TBI and NTBI included codes for fractures (S02, S06), hypoxia (R09.02), anoxia (G91-G93), anaphylaxis (T78), and acute medical clinical incidents (varies).

Results: Within both inpatient and emergency department admissions, the majority of patient visits, both fatal and nonfatal, were reported as "poisoning by any drug" and did not have the presence of a traumatic or non-traumatic brain injury. However, there were a significant number of patients that were admitted with both a "poisoning by any drug and non-traumatic brain injury". Of the reported incidents, there were 14,310 non-fatal and 750 fatal cases that presented with both drug and a non-traumatic brain injury in an inpatient admission and 8,435 non-fatal and 42 fatal cases in the emergency department. In comparison, there were 157 non-fatal and 18 fatal drug and a traumatic brain injury in an inpatient setting and 28 non-fatal and 0 fatal in the emergency department.

Conclusion: The data supports an intersection between drug use and non-traumatic brain injury. These conclusions call for a greater need for prevention strategies aimed at decreasing both drug use and behaviors that result in a non-traumatic brain injury.

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Drug Use and Brain Injury in Kentucky Acute Care Facilities in 2020

Introduction: Individuals who overdose may acquire a brain injury due to hypoxia, and individuals with traumatic brain injury are at greater risk for substance misuse. The purpose of this project was to determine if there is cooccurrence of drug use and overdose and brain injury.

Methods: Using the 2020 Kentucky Outpatient Service and Inpatient Hospitalization Database, encounters for traumatic brain injury (TBI), acquired, non-traumatic brain injury (NTBI), substance use disorder, and overdose were identified using the International Classification of Disease, 10th Edition – Clinical Management. To collect this data, ICD-10-CM codes T36.X-T50.X were used to code for drug use and F11-F19 to code for drug disorder. Coding for TBI and NTBI included codes for fractures (S02, S06), hypoxia (R09.02), anoxia (G91-G93), anaphylaxis (T78), and acute medical clinical incidents (varies).

Results: Within both inpatient and emergency department admissions, the majority of patient visits, both fatal and nonfatal, were reported as "poisoning by any drug" and did not have the presence of a traumatic or non-traumatic brain injury. However, there were a significant number of patients that were admitted with both a "poisoning by any drug and non-traumatic brain injury". Of the reported incidents, there were 14,310 non-fatal and 750 fatal cases that presented with both drug and a non-traumatic brain injury in an inpatient admission and 8,435 non-fatal and 42 fatal cases in the emergency department. In comparison, there were 157 non-fatal and 18 fatal drug and a traumatic brain injury in an inpatient setting and 28 non-fatal and 0 fatal in the emergency department.

Conclusion: The data supports an intersection between drug use and non-traumatic brain injury. These conclusions call for a greater need for prevention strategies aimed at decreasing both drug use and behaviors that result in a non-traumatic brain injury.